Cardiac Left Ventricular Hypertrophy (Record no. 17801)
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000 -LEADER | |
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fixed length control field | 02618nam a22002897a 4500 |
001 - CONTROL NUMBER | |
control field | A. 6271 |
003 - CONTROL NUMBER IDENTIFIER | |
control field | OSt |
005 - DATE AND TIME OF LATEST TRANSACTION | |
control field | 20240305193418.0 |
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION | |
fixed length control field | 220509b |||||||| |||| 00| 0 eng d |
020 ## - INTERNATIONAL STANDARD BOOK NUMBER | |
International Standard Book Number | 0898386128 |
International Standard Book Number | 9780898386127 |
040 ## - CATALOGING SOURCE | |
Transcribing agency | DLC |
082 ## - DEWEY DECIMAL CLASSIFICATION NUMBER | |
Classification number | 616.12 KEU ed |
100 ## - MAIN ENTRY--PERSONAL NAME | |
Personal name | Henk Keurs |
9 (RLIN) | 36979 |
245 ## - TITLE STATEMENT | |
Title | Cardiac Left Ventricular Hypertrophy |
250 ## - EDITION STATEMENT | |
Edition statement | illustrated |
260 ## - PUBLICATION, DISTRIBUTION, ETC. | |
Place of publication, distribution, etc. | Boston |
Name of publisher, distributor, etc. | Springer Netherlands, 1983 Original from the University of Michigan Digitized |
Date of publication, distribution, etc. | 11 Aug 2008 |
300 ## - PHYSICAL DESCRIPTION | |
Extent | 285 pages |
490 ## - SERIES STATEMENT | |
Volume/sequential designation | Volume 33 of Developments in Cardiovascular Medicine, ISSN 0166-9842; Volume 33 of Developments in Nuclear Medicine |
520 ## - SUMMARY, ETC. | |
Summary, etc. | Left ventricular hypertrophy (LVH) is usually considered to be a compen satory adjustment of heart muscle to an inreased work load. LVH develops in the course of valvular or congenital heart disease, or when part of the myocardium is damaged by long-standing ischemia or infarction. In the hypertrophied heart the muscle fibers increase in size, not in number. The fibers are found to contain a larger number of myofibrils and the cell organelles are larger. From epidemiologic studies it is known that even mild LVH is associated with myocardial ischemia, ventricular arrhythmias, and sudden cardiac death. Most cases of LVH show focal degenerative tissue changes including cellular atrophy, myofibrillar disorganization, interstitial fibrosis, and loss of intracellular connections. Myocardial dysfunction develops and, unlike the functional adaptive changes found in pure hypertrophy, is not reversible by surgical treatment of the valvular heart disease or medical correction of hypertension. Interstitial fibrosis, intracellular changes of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden death, a well-recognized problem in patients with a variety of heart diseases. Even When successfully treated, the patients may remain at risk if the compensatory hypertrophy is not fully reversed. Epidemiologic studies conducted in the Framingham population in the early 1950' s demonstrated LVH according to electrocardiographic criteria in 1. 5% of the population; 2% of the population had LVH according to chest X-ray criteria. |
600 ## - SUBJECT ADDED ENTRY--PERSONAL NAME | |
General subdivision | Medical / Clinical Medicine |
9 (RLIN) | 26816 |
General subdivision | Medical / Cardiology |
9 (RLIN) | 26817 |
General subdivision | Medical › Cardiology |
9 (RLIN) | 27540 |
700 ## - ADDED ENTRY--PERSONAL NAME | |
9 (RLIN) | 36980 |
9 (RLIN) | 36981 |
942 ## - ADDED ENTRY ELEMENTS (KOHA) | |
Source of classification or shelving scheme | ddc |
Koha item type | BOOKS |
Withdrawn status | Lost status | Source of classification or shelving scheme | Damaged status | Not for loan | Collection | Home library | Current library | Shelving location | Date acquired | Total checkouts | Full call number | Barcode | Date last seen | Price effective from | Koha item type |
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MWALIMU NYERERE LEARNING RESOURCES CENTRE-CUHAS BUGANDO | MWALIMU NYERERE LEARNING RESOURCES CENTRE-CUHAS BUGANDO | 05/09/2022 | 616.12 KEU ed | A. 6271 | 05/09/2022 | 05/09/2022 | BOOKS |