Cardiac Left Ventricular Hypertrophy (Record no. 17801)

MARC details
000 -LEADER
fixed length control field 02618nam a22002897a 4500
001 - CONTROL NUMBER
control field A. 6271
003 - CONTROL NUMBER IDENTIFIER
control field OSt
005 - DATE AND TIME OF LATEST TRANSACTION
control field 20240305193418.0
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION
fixed length control field 220509b |||||||| |||| 00| 0 eng d
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 0898386128
International Standard Book Number 9780898386127
040 ## - CATALOGING SOURCE
Transcribing agency DLC
082 ## - DEWEY DECIMAL CLASSIFICATION NUMBER
Classification number 616.12 KEU ed
100 ## - MAIN ENTRY--PERSONAL NAME
Personal name Henk Keurs
9 (RLIN) 36979
245 ## - TITLE STATEMENT
Title Cardiac Left Ventricular Hypertrophy
250 ## - EDITION STATEMENT
Edition statement illustrated
260 ## - PUBLICATION, DISTRIBUTION, ETC.
Place of publication, distribution, etc. Boston
Name of publisher, distributor, etc. Springer Netherlands, 1983 Original from the University of Michigan Digitized
Date of publication, distribution, etc. 11 Aug 2008
300 ## - PHYSICAL DESCRIPTION
Extent 285 pages
490 ## - SERIES STATEMENT
Volume/sequential designation Volume 33 of Developments in Cardiovascular Medicine, ISSN 0166-9842; Volume 33 of Developments in Nuclear Medicine
520 ## - SUMMARY, ETC.
Summary, etc. Left ventricular hypertrophy (LVH) is usually considered to be a compen satory adjustment of heart muscle to an inreased work load. LVH develops in the course of valvular or congenital heart disease, or when part of the myocardium is damaged by long-standing ischemia or infarction. In the hypertrophied heart the muscle fibers increase in size, not in number. The fibers are found to contain a larger number of myofibrils and the cell organelles are larger. From epidemiologic studies it is known that even mild LVH is associated with myocardial ischemia, ventricular arrhythmias, and sudden cardiac death. Most cases of LVH show focal degenerative tissue changes including cellular atrophy, myofibrillar disorganization, interstitial fibrosis, and loss of intracellular connections. Myocardial dysfunction develops and, unlike the functional adaptive changes found in pure hypertrophy, is not reversible by surgical treatment of the valvular heart disease or medical correction of hypertension. Interstitial fibrosis, intracellular changes of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden death, a well-recognized problem in patients with a variety of heart diseases. Even When successfully treated, the patients may remain at risk if the compensatory hypertrophy is not fully reversed. Epidemiologic studies conducted in the Framingham population in the early 1950' s demonstrated LVH according to electrocardiographic criteria in 1. 5% of the population; 2% of the population had LVH according to chest X-ray criteria.
600 ## - SUBJECT ADDED ENTRY--PERSONAL NAME
General subdivision Medical / Clinical Medicine
9 (RLIN) 26816
General subdivision Medical / Cardiology
9 (RLIN) 26817
General subdivision Medical › Cardiology
9 (RLIN) 27540
700 ## - ADDED ENTRY--PERSONAL NAME
9 (RLIN) 36980
9 (RLIN) 36981
942 ## - ADDED ENTRY ELEMENTS (KOHA)
Source of classification or shelving scheme ddc
Koha item type BOOKS
Holdings
Withdrawn status Lost status Source of classification or shelving scheme Damaged status Not for loan Collection Home library Current library Shelving location Date acquired Total checkouts Full call number Barcode Date last seen Price effective from Koha item type
            MWALIMU NYERERE LEARNING RESOURCES CENTRE-CUHAS BUGANDO MWALIMU NYERERE LEARNING RESOURCES CENTRE-CUHAS BUGANDO   05/09/2022   616.12 KEU ed A. 6271 05/09/2022 05/09/2022 BOOKS
Catholic University of  Health and Allied Sciences - CUHAS
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